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Significant loading dose of clopidogrel is struggling to satisfactorily inhibit platelet reactivity in individuals with glycoprotein IIIA gene polymorphism: a genetic substudy of PRAGUE-8 trial. Blood Coagul Fibrinolysis
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Inside the context of PAR4 activation, SKI-606 diminished platelet spreading in both equally genotypes, While substantial genotype distinctions have been nevertheless noticed. These knowledge advise that Src signaling in KI platelets brings about enhanced platelet spreading Which inhibition of Src is ample to normalize platelet spreading to wild-form concentrations.
These data indicate that a number of signaling pathways downstream from PAR4 activation, outside of Src, modify platelet attachment from the context with the Pro32Pro33 mutation.
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